Jeff Hansen

Jeff Hansen

| This email address is being protected from spambots. You need JavaScript enabled to view it.

Research Editor

jeffhans@uab.edu | (205) 209-2355

Communicates UAB research discoveries and initiatives from across the university for a variety of audiences.

Specific beats: 

  • Alabama Drug Discovery Alliance
  • Biochemistry and Molecular Genetics 
  • Biomatrix Engineering and Regenerative Medicine 
  • Cell biology 
  • Center for Biophysical Sciences and Engineering 
  • CCTS
  • Center for Metabolic Bone Disease 
  • Microbiology 
  • Neurobiology 
  • Comprehensive Neuroscience Center 
  • Pathology, research shared with MS2
  • Pharmacology and Toxicology 
  • Physiology and Biophysics 
  • UAB Research Foundation/IIE 
  • Research Administration

AdCOVID stimulated both strong serum neutralizing activity and potent mucosal IgA immunity in the respiratory tract.

Two treatments given together before birth —  magnesium sulfate and corticosteroids — can improve outcomes in preterm children.

This work is a step forward in understanding early molecular changes that influence the development of addiction, and may have application to the role of similar gene programs that mediate other types of behavior, memory formation or neuropsychiatric disorders.

This study was done in mice and with a novel, tissue-engineered, three-dimensional breast cancer mimetic system.
This mouse study looked at a group of immune cells called innate-like B-1 B cells.
FASEB is the largest coalition of biological and biomedical research associations in the United States.
Injections of two chemicals in a slow-release form significantly reduced the size of dead heart tissue and improved the function of the left ventricle.
This novel finding will enable experimental studies to determine whether and how these microbes play a role in triggering the disease.
A mouse model and previous studies suggest that genetic intervention in SHANK3-related ASD may be most effective earlier in development.
Results show that retinitis pigmentosa 59 may not be a congenital disorder of glycosylation, as was long assumed.
Page 21 of 52