Jeff Hansen

Jeff Hansen

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Research Editor

jeffhans@uab.edu | (205) 209-2355

Communicates UAB research discoveries and initiatives from across the university for a variety of audiences.

Specific beats: 

  • Alabama Drug Discovery Alliance
  • Biochemistry and Molecular Genetics 
  • Biomatrix Engineering and Regenerative Medicine 
  • Cell biology 
  • Center for Biophysical Sciences and Engineering 
  • CCTS
  • Center for Metabolic Bone Disease 
  • Microbiology 
  • Neurobiology 
  • Comprehensive Neuroscience Center 
  • Pathology, research shared with MS2
  • Pharmacology and Toxicology 
  • Physiology and Biophysics 
  • UAB Research Foundation/IIE 
  • Research Administration
The biomarker, the protein kinase LRRK2, is a promising candidate for future exploration.
A survival advantage for female humans stands out in a UAB review of sex differences in longevity across many species. Understanding why could inform treatments to extend healthy lifespans.
A robotic 3-D printer will build the tissue through computer-assisted design and computer-assisted manufacturing, but using living cells as the pieces of the structure.
This competitive award recognizes outstanding achievements and contributions to VA research and provides five years of salary support.
The UAB course was part of a master’s degree program in bioinformatics for researchers in Mali, where malaria is a scourge.
Cystic fibrosis lung cells were restored to 50 percent of healthy function in work that provides the first evidence that novel therapeutic strategies for human patients can be identified based on yeast studies.
UAB researchers will use a $1.8 million grant to look at single cells for altered expression of the interferon gamma receptor gene.
VA Research support for UAB clinicians and scientists has been important for six decades.
Results show that JAK/STAT pathway inhibitors may be a new class of therapeutic treatments for patients with Parkinson’s disease. Acting by reducing inflammation, they prevent neurodegeneration in animal models and may be an important new approach to slow progression of the disease.

Researchers have proposed a model that resolves a seeming paradox in one of the most intriguing areas of the brain, exploring how immature granule cells in the dentate gyrus appear able to enhance pattern separation due to lesser synaptic connectivity than mature cells.

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